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c-Kit dysfunction impairs myocardial healing after infarction
Massimo Cimini
, Shafie Fazel
,
Sun Zhuo
, Munira Xaymardan
, Hiroko Fujii
, Richard D. Weisel
, Ren Ke Li
Research output
:
Contribution to journal
›
Article
›
peer-review
67
Link opens in a new tab
Scopus citations
Overview
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Dive into the research topics of 'c-Kit dysfunction impairs myocardial healing after infarction'. Together they form a unique fingerprint.
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Keyphrases
Infarction
100%
C-kit
100%
Myocardial Healing
100%
Mutant Mice
66%
Myocardial Infarction
50%
α-smooth muscle Actin (α-SMA)
50%
Infarct Expansion
50%
Bone Marrow
33%
Blood Vessels
33%
Infarcted Heart
33%
CD31
33%
Non-vascular
33%
Transplantation
16%
Flow Cytometry
16%
Healing
16%
Cardiac Function
16%
Receptor Function
16%
Bone Marrow Cells
16%
Microscopy
16%
Infarct
16%
Myofibroblast
16%
Day 14
16%
Fold Increase
16%
Infiltration
16%
Littermate
16%
Chimeric Mice
16%
Coronary Ligation
16%
Cardiac Repair
16%
Cardiac Remodeling
16%
Repair Tissue
16%
Ventricular Dilation
16%
Endogenous Repair
16%
Immunology and Microbiology
Mouse Mutant
100%
Smooth Muscle
75%
Wild Type
75%
Actin
75%
Blood Vessels
50%
CD31
50%
Mouse
25%
Heart Function
25%
Bone Marrow Cell
25%
Myofibroblast
25%
Tissue Repair
25%
Stem Cell Factor Receptor
25%
Pharmacology, Toxicology and Pharmaceutical Science
Infarction
100%
Mouse Mutant
80%
Heart Infarction
60%
Smooth Muscle Actin
60%
Mouse
20%
Flow Cytometry
20%
Tissue Repair
20%
Heart Ventricle Remodeling
20%
Patient History of Transplantation
20%
Stem Cell Factor Receptor
20%
Medicine and Dentistry
Infarction
100%
Myocardial Infarction
60%
Smooth Muscle Actin
60%
Heart Function
20%
Bone Marrow Cell
20%
Flow Cytometry
20%
Myofibroblast
20%
Tissue Repair
20%
Patient History of Transplantation
20%
Heart Ventricle Remodeling
20%
Stem Cell Factor Receptor
20%
Coronary Artery Ligation
20%