Chronic Myelogenous Leukemia

Alfonso Quintás-Cardama; Jorge Cortes; Hagop Kantarjian; Susan O'Brien

doi:10.1002/9781444318531.ch7

Chronic Myelogenous Leukemia

Alfonso Quintás-Cardama, Jorge Cortes, Hagop Kantarjian, Susan O'Brien

Research output: Chapter in Book/Report/Conference proceeding › Chapter

Original language	English (US)
Title of host publication	Molecular Hematology
Subtitle of host publication	Third Edition
Publisher	Wiley-Blackwell
Pages	76-88
Number of pages	13
ISBN (Print)	9781405182317
DOIs	https://doi.org/10.1002/9781444318531.ch7
State	Published - Mar 10 2010
Externally published	Yes

Keywords

BCR-ABL 1 kinase signaling pathways
BP CML, characterized by prominent hypercatabolic symptoms
Chronic myelogenous leukemia
Chronic myeloid leukemia (CML), clonal myeloproliferative neoplasm from pluripotent stem cell
Emergence of imatinib mesylate -change in therapeutic algorithm for CML
Genome-wide analyses of gene expression profiles -prognostication tools in multiple malignancies
Imatinib resistance -categorized as primary (also "refractoriness")
Signaling pathways activated by BCR-ABL1 kinase
Stem cell transplantation -allogeneic SCT, therapeutic modality for patients with human leukocyte antigen (HLA)-identical siblings

ASJC Scopus subject areas

General Medicine

UN SDGs

This output contributes to the following UN Sustainable Development Goals (SDGs)

Access to Document

10.1002/9781444318531.ch7

Cite this

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title = "Chronic Myelogenous Leukemia",

keywords = "BCR-ABL 1 kinase signaling pathways, BP CML, characterized by prominent hypercatabolic symptoms, Chronic myelogenous leukemia, Chronic myeloid leukemia (CML), clonal myeloproliferative neoplasm from pluripotent stem cell, Emergence of imatinib mesylate -change in therapeutic algorithm for CML, Genome-wide analyses of gene expression profiles -prognostication tools in multiple malignancies, Imatinib resistance -categorized as primary (also {"}refractoriness{"}), Signaling pathways activated by BCR-ABL1 kinase, Stem cell transplantation -allogeneic SCT, therapeutic modality for patients with human leukocyte antigen (HLA)-identical siblings",

author = "Alfonso Quint{\'a}s-Cardama and Jorge Cortes and Hagop Kantarjian and Susan O'Brien",

year = "2010",

month = mar,

day = "10",

doi = "10.1002/9781444318531.ch7",

language = "English (US)",

isbn = "9781405182317",

pages = "76--88",

booktitle = "Molecular Hematology",

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}

TY - CHAP

T1 - Chronic Myelogenous Leukemia

AU - Quintás-Cardama, Alfonso

AU - Cortes, Jorge

AU - Kantarjian, Hagop

AU - O'Brien, Susan

PY - 2010/3/10

Y1 - 2010/3/10

KW - BCR-ABL 1 kinase signaling pathways

KW - BP CML, characterized by prominent hypercatabolic symptoms

KW - Chronic myelogenous leukemia

KW - Chronic myeloid leukemia (CML), clonal myeloproliferative neoplasm from pluripotent stem cell

KW - Emergence of imatinib mesylate -change in therapeutic algorithm for CML

KW - Genome-wide analyses of gene expression profiles -prognostication tools in multiple malignancies

KW - Imatinib resistance -categorized as primary (also "refractoriness")

KW - Signaling pathways activated by BCR-ABL1 kinase

KW - Stem cell transplantation -allogeneic SCT, therapeutic modality for patients with human leukocyte antigen (HLA)-identical siblings

UR - http://www.scopus.com/inward/record.url?scp=84885786072&partnerID=8YFLogxK

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U2 - 10.1002/9781444318531.ch7

DO - 10.1002/9781444318531.ch7

M3 - Chapter

AN - SCOPUS:84885786072

SN - 9781405182317

SP - 76

EP - 88

BT - Molecular Hematology

PB - Wiley-Blackwell

ER -

Chronic Myelogenous Leukemia

Keywords

ASJC Scopus subject areas

UN SDGs

Access to Document

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Cite this