Disruption of inducible 6-phosphofructo-2-kinase impairs the suppressive effect of PPARγ activation on diet-induced intestine inflammatory response

Keyphrases

• 1-factor 9%
• 6-phosphofructo-2-kinase 100%
• Adipocyte 9%
• Adipose Tissue 9%
• Antidiabetic Effect 9%
• C-Jun N-terminal Kinase 9%
• Diet-induced 100%
• Glycolysis 9%
• High-fat Diet 36%
• Homeostasis Model Assessment of Insulin Resistance (HOMA-IR) 9%
• Inflammatory Cytokine Response 9%
• Inflammatory Response 100%
• Insulin Sensitivity 9%
• Insulin Sensitizers 9%
• Intestine 100%
• Littermate 9%
• Low-fat Diet 9%
• Metabolic Response 9%
• Mouse Intestine 9%
• Nuclear factor-κB 9%
• Peroxisome Proliferator-activated Receptor 100%
• PFKFB3 100%
• Phosphorylation 9%
• Regulatory Enzymes 9%
• Rosiglitazone 9%
• Suppression Effect 100%
• Toll-like 9%
• Undernutrition 18%
• Wild Mice 18%

Immunology and Microbiology

• Adipocyte 12%
• Adipose Tissue 12%
• Agonist 12%
• Amino Terminal Sequence 12%
• Antidiabetic Activity 12%
• Homeostasis 12%
• Immunoglobulin Enhancer Binding Protein 12%
• Inflammation Response 100%
• Insulin Resistance 12%
• Insulin Sensitivity 12%
• Intestine 100%
• Mouse 75%
• Peroxisome 100%
• Proinflammatory Cytokine 12%
• TLR4 12%
• Wild Type 12%
• Wild Type Mouse 25%