Increased long chain acyl CoA in duchenne muscular dystrophy

James E. Carroll, Aida Villadiego, Michael H. Brooke

Research output: Contribution to journalArticlepeer-review

17 Scopus citations


Compared with normal or denervated human muscle, long chain acyl CoA was increased in muscle from patients with Duchenne dystrophy. Free and short chain acylcarnitine were reduced in Duchenne muscle, whereas long chain acylcarnitine was preserved. The accumulation of long chain fatty acid derivatives may imply disruption of fatty acid oxidation.

Original languageEnglish (US)
Pages (from-to)1507-1510
Number of pages4
Issue number11
StatePublished - Nov 1983
Externally publishedYes

ASJC Scopus subject areas

  • Clinical Neurology


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