Oxidative phenotype protects myofibers from pathological insults induced by chronic heart failure in mice

Keyphrases

• Insults 100%
• Chronic Heart Failure 100%
• Myofiber 100%
• Oxidative Phenotype 100%
• Glycolytic 60%
• Exercise Intolerance 40%
• Plantaris muscle 40%
• Transgenic Mice 20%
• Protein Expression 20%
• Overexpression 20%
• Cross-sectional Area 20%
• Skeletal muscle 20%
• Sarcomere 20%
• Coactivator 20%
• Peroxisome Proliferator-activated Receptor 20%
• Muscle Mass 20%
• Atrophy 20%
• Tibialis Anterior muscle 20%
• Cachexia 20%
• Lung Weight 20%
• Cytochrome c Oxidase 20%
• Skeletal Disorders 20%
• Fractional Shortening 20%
• Soleus muscle 20%
• Calsequestrin 20%
• Muscle Fiber Composition 20%
• Running Distance 20%
• Vastus Lateralis muscle 20%
• Atrogin-1 20%
• MAFbx 20%
• Fiber-type Switching 20%

Medicine and Dentistry

• Heart Failure 100%
• Skeletal Muscle 40%
• Exercise Intolerance 40%
• Plantaris Muscle 40%
• Protein Expression 20%
• Sarcomere 20%
• Body Weight 20%
• Cachexia 20%
• Transgenic Mouse 20%
• Cytochrome C Oxidase 20%
• Peroxisome Proliferator Activated Receptor 20%
• Calsequestrin 20%
• Soleus Muscle 20%
• Tibialis Anterior Muscle 20%
• Vastus Lateralis Muscle 20%
• Atrogin 1 20%

Pharmacology, Toxicology and Pharmaceutical Science

• Mouse 100%
• Heart Failure 100%
• Peroxisome Proliferator Activated Receptor 20%
• Cachexia 20%
• Transgenic Mouse 20%
• Cytochrome C Oxidase 20%
• Albendazole 20%
• Calsequestrin 20%
• Atrogin 1 20%