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Expression, genomic structure and high resolution mapping to 19p13.2 of the human smooth muscle cell calponin gene

  • Joseph M. Miano(corresponding author)
    ,
  • Ralf Krahe
    ,
  • Emilio Garcia
    ,
  • Jeffrey M. Elliott
    ,
  • Eric N. Olson
*Corresponding author for this work
  • Medical College of Wisconsin
    ,
  • University of Helsinki
    ,
  • Lawrence Livermore National Laboratory
    ,
  • University of Texas at Dallas
Scholary Output:
Contribution to journal
Article
Peer-review

Sustainable Development Goals

  • SDG 3 - Good Health and Well-being
    SDG 3 Good Health and Well

Abstract

Smooth muscle cells (SMC) express a battery of cell-restricted differentiation genes, many of which are down-regulated during the course of vascular disease. Here, we present the mRNA expression, genomic structure and chromosomal mapping of the gene encoding human smooth muscle cell calponin (SMCC). Human SMCC transcripts are restricted to tissues and cells of SMC origin and, in the latter case, appear to be uniquely controlled in two distinct human SMC lines of uterine and aortic origin. Restriction mapping, Southern blot and PCR analysis of a 70-kb human bacterial artificial chromosome (BAC) revealed a genomic structure (seven exons spanning > 11 kb) very similar to that reported for the mouse SMCC gene. Using a variety of human-rodent somatic cell hybrid and radiation hybrid mapping panels, the human SMCC gene was mapped to a genomic interval of less than 1.32 Mb in 19p13.2. These results provide new information concerning the regulation of SMCC gene expression and demonstrate the utility of two human SMC lines for the further characterization of this gene's expression control. The identification of a BAC harboring the entire human SMCC locus represents an important reagent for future analysis of SMCC regulatory sequences. Finally, the localization of SMCC to a defined genomic interval will facilitate an analysis of its potential as a candidate gene for disease phenotypes mapping to 19p13.2.

Publication Information

Output type

Scholary Output:
Contribution to journal
Article
Peer-review

Original language

English (US)

Pages from-to (Number of pages)

Pages 215-224 (10 pages)

Journal (Volume, Issue Number)

Gene (Volume 197, Issue 1-2)

Publication milestones

  • Published - 09/15/1997

Publication status

Published - 09/15/1997

ISSN

0378-1119

Publication IDs

  • Scopus: 0030880199
  • PubMed: 9332369

Publication metrics

Metrics

Scopus
citations
Fractional count
1
Fractional count
0.20
Fractional count
4
Fractional count
0.80
Fractional count
1
Fractional count
1
SciVal
FWCI
0.88
SciVal
Author count
5
SciVal
citations
18
SciVal
Paper percentile
70

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Captures
7
Citation count
21
Mentions
1

Funding Details

The authors thank Dr Michael J. Siciliano (Department of Molecular Genetics, University of Texas MD Anderson Cancer Center) and Drs Grace Hobson and Geeta Iyers (A.I. DuPont Institute, Wilmington, DE) for kindly providing the DNAs for mapping panel 1, the mono-chromosomal and human chromosome 19 regional mapping panels, and mapping panel 2, respectively. They also thank Dr James McDougall (Fred Hutchinson Cancer Center) for furnishing the human aortic and uterine SMC lines, Dr Kirkwood A. Pritchard, Jr (Medical College of Wisconsin) for the HUVEC, and Dr John Fenton (New York State Department of Health) for the generous supply of α -thrombin. The graphical assistance of Alisha Tizenor and Louise Kelly-Roloff is also greatly appreciated. This work was supported by grants from the National Institutes of Health, the Muscular Dystrophy Association and The Robert A. Welch Foundation to ENO. JMM is supported by an American Heart Association Grant-in-Aid (Wisconsin Affiliate), and RK, in the laboratory of Dr Michael J. Siciliano, was supported by the Muscular Dystrophy Association. The work carried out at Lawrence Livermore National Laboratory was performed under the auspices of the US Department of Energy, contract no. W-7405-ENG-48.