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Gain-of-function of progesterone receptor membrane component 2 ameliorates ischemic brain injury

  • Chao Zhou
    ,
  • Taiyang Zhu
    ,
  • Wanyan Ni
    ,
  • Hui Zhou
    ,
  • Jiaxing Song
    ,
  • Miao Wang
*Corresponding author for this work
  • Xuzhou Medical University
    ,
  • Army Medical University
    ,
Scholary Output:
Contribution to journal
Article
Peer-review

Open access

Sustainable Development Goals

  • SDG 3 - Good Health and Well-being
    SDG 3 Good Health and Well

Abstract

Objective: Progesterone receptor membrane component 2 (PGRMC2) belongs to the membrane-associated progesterone receptor family, which regulates multiple pathophysiological processes. However, the role of PGRMC2 in ischemic stroke remains unexplored. The present study sought to determine the regulatory role of PGRMC2 in ischemic stroke. Methods: Male C57BL/6J mice were subjected to middle cerebral artery occlusion (MCAO). The protein expression level and localization of PGRMC2 were examined by western blotting and immunofluorescence staining. The gain-of-function ligand of PGRMC2 (CPAG-1, 45 mg/kg) was intraperitoneally injected into sham/MCAO mice, and brain infarction, blood–brain barrier (BBB) leakage, and sensorimotor functions were evaluated by magnetic resonance imaging, brain water content, Evans blue extravasation, immunofluorescence staining, and neurobehavioral tests. The astrocyte and microglial activation, neuronal functions, and gene expression profiles were revealed by RNA sequencing, qPCR, western blotting, and immunofluorescence staining after surgery and CPAG-1 treatment. Results: Progesterone receptor membrane component 2 was elevated in different brain cells after ischemic stroke. Intraperitoneal delivery of CPAG-1 reduced infarct size, brain edema, BBB leakage, astrocyte and microglial activation, and neuronal death, and improved sensorimotor deficits after ischemic stroke. Conclusion: CPAG-1 acts as a novel neuroprotective compound that could reduce neuropathologic damage and improve functional recovery after ischemic stroke.

Publication Information

Output type

Scholary Output:
Contribution to journal
Article
Peer-review

Original language

English (US)

Pages from-to (Number of pages)

Pages 1585-1601 (17 pages)

Journal (Volume, Issue Number)

CNS Neuroscience and Therapeutics (Volume 29, Issue 6)

Publication milestones

  • Published - 06/2023

Publication status

Published - 06/2023

ISSN

1755-5930

Publication IDs

  • Scopus: 85148340809
  • PubMed: 36794556

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Funding Details

This work was supported by the National Nature Science Foundation of China to FH (81571469 and 82171420).
FunderFunding numbers
NSFC
81571469, 82171420