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Predictors of mortality in patients with hereditary hemorrhagic telangiectasia

  • the Brain Vascular Malformation Consortium HHT Investigator Group
    ,
  • K. P. Thompson
    ,
  • J. Nelson
    ,
  • H. Kim
    ,
  • L. Pawlikowska
    ,
  • D. A. Marchuk
*Corresponding author for this work
  • University of Toronto
    ,
  • University of California at San Francisco
    ,
  • Duke University
    ,
  • St. Joseph's Hospital and Medical Center, Phoenix
    ,
Scholary Output:
Contribution to journal
Article
Peer-review

Open access

Sustainable Development Goals

  • SDG 3 - Good Health and Well-being
    SDG 3 Good Health and Well

Abstract

Background: Retrospective questionnaire and healthcare administrative data suggest reduced life expectancy in untreated hereditary hemorrhagic telangiectasia (HHT). Prospective data suggests similar mortality, to the general population, in Denmark’s centre-treated HHT patients. However, clinical phenotypes vary widely in HHT, likely affecting mortality. We aimed to measure predictors of mortality among centre-treated HHT patients. HHT patients were recruited at 14 HHT centres of the Brain Vascular Malformation Consortium (BVMC) since 2010 and followed annually. Vital status, organ vascular malformations (VMs) and clinical symptoms data were collected at baseline and during follow-up (N = 1286). We tested whether organ VMs, HHT symptoms and HHT genes were associated with increased mortality using Cox regression analysis, adjusting for patient age, sex, and smoking status. Results: 59 deaths occurred over average follow-up time of 3.4 years (max 8.6 years). A history of anemia was associated with increased mortality (HR = 2.93, 95% CI 1.37–6.26, p = 0.006), as were gastro-intestinal (GI) bleeding (HR = 2.63, 95% CI 1.46–4.74, p = 0.001), and symptomatic liver VMs (HR = 2.10, 95% CI 1.15–3.84, p = 0.015). Brain VMs and pulmonary arteriovenous malformations (AVMs) were not associated with mortality (p > 0.05). Patients with SMAD4 mutation had significantly higher mortality (HR = 18.36, 95% CI 5.60–60.20, p < 0.001) compared to patients with ACVRL1 or ENG mutation, but this estimate is imprecise given the rarity of SMAD4 patients (n = 33, 4 deaths). Conclusions: Chronic GI bleeding, anemia and symptomatic liver VMs are associated with increased mortality in HHT patients, independent of age, and in keeping with the limited treatment options for these aspects of HHT. Conversely, mortality does not appear to be associated with pulmonary AVMs or brain VMs, for which patients are routinely screened and treated preventatively at HHT Centres. This demonstrates the need for development of new therapies to treat chronic anemia, GI bleeding, and symptomatic liver VMs in order to reduce mortality among HHT patients.

Publication Information

Output type

Scholary Output:
Contribution to journal
Article
Peer-review

Original language

English (US)

Article number

12

Journal (Volume, Issue Number)

Orphanet Journal of Rare Diseases (Volume 16, Issue 1)

Publication milestones

  • Published - 12/2021

Publication status

Published - 12/2021

ISSN

1750-1172

Publication IDs

  • Scopus: 85098957922
  • PubMed: 33407668

Publication metrics

Metrics

Scopus
citations
SciVal
Author count
30
SciVal
Paper percentile
81
Fractional count
1
Fractional count
0.03
Fractional count
28
Fractional count
0.97
Fractional count
1
Fractional count
1

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Social media
34
Captures
23
Citation count
23

Funding Details

The Brain Vascular Malformation Consortium (U54NS065705) is a part of the NCATS Rare Diseases Clinical Research Network (RDCRN) and is supported by the RDCRN Data Management and Coordinating Center (DMCC) (U2CTR002818). RDCRN is an initiative of the Office of Rare Diseases Research (ORDR), NCATS, funded through a collaboration between NCATS and NINDS. M.E.F. was also supported by the Nelson Arthur Hyland Foundation and Li Ka Shing Knowledge Institute.
FundersFunding numbers
DMCC
U2CTR002818
Nelson Arthur Hyland Foundation
-
NINDS
U54NS065705
RDCRN
-
LKSIV
-