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Targeting the tumor vasculature with combretastatin A-4 disodium phosphate: Effects on radiation therapy

  • Lingyun Li
    ,
  • Amyn Rojiani
    ,
  • Dietmar W. Siemann(corresponding author)
*Corresponding author for this work
  • University of Florida
    ,
  • University of South Florida
Scholary Output:
Contribution to journal
Article
Peer-review

Sustainable Development Goals

  • SDG 3 - Good Health and Well-being
    SDG 3 Good Health and Well

Abstract

Purpose: The aim of this study was to evaluate the antitumor efficacy of combretastatin A-4 disodium phosphate (combretastatin prodrug) in the rodent KHT sarcoma model either alone or in combination with radiation therapy. Methods: KHT tumors were grown in C3H/HeJ mice. Combretastatin A-4 prodrug was injected intraperitoneally at doses ranging from 10 to 100 mg/kg. Tumors were irradiated in unanesthetized mice using a 137Cs source. Tumor response to combretastatin A-4 prodrug was assessed by histological evaluations as well as an in vivo to in vitro cell survival assay. Results: Histological evaluation showed morphological damage of tumor cells within a few hours after drug treatment, followed by extensive central necrosis. Administering increasing doses of combretastatin A-4 prodrug to tumor- bearing mice resulted in a dose-dependent increase in cell killing irrespective of whether the tumors were irradiated or not. When combined with radiation, a 100 mg/kg dose of combretastatin A-4 prodrug reduced tumor cell survival 10-500-fold lower than that seen with radiation alone. Further, the shape of the cell survival curve observed following the combination therapy suggested that including combretastatin in the treatment had a major effect on the radiation-resistant hypoxic cell subpopulation associated with this tumor. Conclusion: The present results demonstrated that in the KHT sarcoma, combretastatin A-4 prodrug caused rapid vascular shutdown, a concentration- dependent direct cell killing, and effective enhancement of the antitumor effects of radiation therapy.

Publication Information

Output type

Scholary Output:
Contribution to journal
Article
Peer-review

Original language

English (US)

Pages from-to (Number of pages)

Pages 899-903 (5 pages)

Journal (Volume, Issue Number)

International Journal of Radiation Oncology Biology Physics (Volume 42, Issue 4)

Publication milestones

  • Published - 11/01/1998

Publication status

Published - 11/01/1998

ISSN

0360-3016

Publication IDs

  • Scopus: 0031742231
  • PubMed: 9845118

Publication metrics

Metrics

Scopus
citations
Fractional count
1
Fractional count
0.33
Fractional count
2
Fractional count
0.67
Fractional count
1
Fractional count
1
SciVal
FWCI
12.70
SciVal
Author count
3
SciVal
citations
130
SciVal
Paper percentile
96
SciVal
Top percentile
5

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Citation count
134
Captures
21

Funding Details

The authors thank Sharon Lepler and David Bloom for providing excellent technical assistance. This work was supported in part by a grant from OXiGENE Inc. and PHS Grant CA 36858.
FundersFunding number
OXiGENE Inc.
-
PHS
-
NCI
R01CA036858