DNA damage response in renal ischemia-reperfusion and ATP-depletion injury of renal tubular cells

Keyphrases

• Renal Tubular Epithelial Cells 100%
• Renal Ischemia Reperfusion 100%
• DNA Damage Response 100%
• ATP Depletion 100%
• Apoptosis 33%
• Phosphorylation 33%
• N-acetylcysteine 25%
• Chk2 25%
• H2AX 25%
• DNA Damage 16%
• Acute Kidney Injury 16%
• Tubular Cell Damage 16%
• Nuclear Foci 16%
• Focus Formation 16%
• KU-55933 16%
• Oxidative Stress 8%
• B-cell Lymphoma 2 (Bcl-2) 8%
• Cell Death 8%
• Stress-related 8%
• Caspase Inhibitor 8%
• Overexpression 8%
• Antioxidant 8%
• Atrophy 8%
• Reperfusion 8%
• Interstitial Fibrosis 8%
• Kidney Tissue 8%
• Tubular Cells 8%
• Ischemic Kidney Injury 8%
• Glycolysis 8%
• Tubular Injury 8%
• In Vivo Depletion 8%
• ATM Inhibitor 8%
• H2AX Phosphorylation 8%
• Tubular Repair 8%
• ATM Phosphorylation 8%

Pharmacology, Toxicology and Pharmaceutical Science

• Injury 100%
• Adenosine Triphosphate 100%
• Kidney Ischemia 100%
• Protein P53 50%
• Acetylcysteine 50%
• Acute Kidney Failure 50%
• 2 Morpholino 6 (1 Thianthrenyl) 4 Pyranone 33%
• Mouse 16%
• Antioxidant 16%
• Caspase Inhibitor 16%
• Cytoprotective Agent 16%
• Fibrosing Alveolitis 16%