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Mechanisms of ionomycin-induced endothelial cell barrier dysfunction
Joe G.N. Garcia
, Kane L. Schaphorst
, Shu Shi
,
Alexander D. Verin
, C. Michael Hart
, Karleen S. Callahan
, Carolyn E. Patterson
Research output
:
Contribution to journal
›
Article
›
peer-review
88
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Scopus citations
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Neuroscience
Kinase
100%
Barrier Cells
100%
Receptor Tyrosine Kinase
66%
Tyrosine Kinase
66%
Protein Kinase A
66%
Thrombin
66%
Focal Adhesion Kinase 1
66%
Myosin Light Chain
66%
Nitric Oxide Synthase
33%
Serine
33%
Threonine
33%
Cyclic Adenosine Monophosphate
33%
Adenylyl Cyclase
33%
Cell Membrane Permeability
33%
Myosin Light-Chain Kinase
33%
Phospholipase A2
33%
Phosphotyrosine
33%
Ionophore
33%
Phosphatase
33%
Calmodulin
33%
Albumin
33%
phosphorylation
33%
Keyphrases
Barrier Dysfunction
100%
Induced Endothelial Cells
100%
Ionomycin
100%
Epithelial Barrier
100%
Endothelial Cells
66%
Protein Kinase
33%
Focal Adhesion Kinase
33%
Myosin Phosphatase
33%
Tyrosine Kinase Activity
33%
Thrombin
33%
CYP125
33%
Concentration-dependent
16%
Kinase Activation
16%
Ca2+ Ionophore
16%
Adenylate Cyclase
16%
Ca2+ Dependence
16%
Endothelial Monolayer
16%
Myosin Light Chain Kinase
16%
Phosphotyrosine
16%
Cell Contraction
16%
Time-dependent Inhibition
16%
Cyclic Adenosine Monophosphate (cAMP)
16%
Albumin
16%
Gap Formation
16%
Electrical Resistance
16%
Cell Permeability
16%
Cell Gap
16%
Phospholipase A2 Activity
16%
Ca2+/calmodulin
16%
Endothelial Permeability
16%
Nitric Oxide Synthase
16%
Serine/threonine Phosphatase
16%
Kinase-independent
16%