Abstract
Ablating mice of Neto1, a component that stabilizes a postsynaptic glutamate receptor, results in a deficit in memory and learning - a deficit ameliorated by administration of a glutamate-receptor stimulant.
Original language | English (US) |
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Pages (from-to) | 302 |
Number of pages | 1 |
Journal | New England Journal of Medicine |
Volume | 361 |
Issue number | 3 |
DOIs | |
State | Published - Jul 16 2009 |
ASJC Scopus subject areas
- Medicine(all)