TY - JOUR
T1 - Unilateral hyperhidrosis after cerebral infarction
AU - Labar, Douglas R.
AU - Mohr, J. P.
AU - Nichols, Fenwick T.
AU - Tatemichi, Thomas K.
PY - 1988/11
Y1 - 1988/11
N2 - Unilateral hyperhidrosis occurred contralateral to acute cerebral infarctions in six patients. Two patients had localized infarctions of opercular cortex, while the other four had large strokes involving both superficial cortical and deep subcortical structures. The unilateral hyperhidrosis typically involved the face and arm and was transient, lasting 1 to 3 days. No other autonomic dysfunction occurred. One patient died, and the others had significant residual neurologic disability, thus indicating poor prognosis when this sign is present. Disruption of a pathway of cortical origin, inhibitory to contralateral sweating, is a proposed mechanism for the hyperhidrosis seen contralateral to acute cerebral infarction in these patients.
AB - Unilateral hyperhidrosis occurred contralateral to acute cerebral infarctions in six patients. Two patients had localized infarctions of opercular cortex, while the other four had large strokes involving both superficial cortical and deep subcortical structures. The unilateral hyperhidrosis typically involved the face and arm and was transient, lasting 1 to 3 days. No other autonomic dysfunction occurred. One patient died, and the others had significant residual neurologic disability, thus indicating poor prognosis when this sign is present. Disruption of a pathway of cortical origin, inhibitory to contralateral sweating, is a proposed mechanism for the hyperhidrosis seen contralateral to acute cerebral infarction in these patients.
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U2 - 10.1212/wnl.38.11.1679
DO - 10.1212/wnl.38.11.1679
M3 - Article
C2 - 3185901
AN - SCOPUS:0023697555
SN - 0028-3878
VL - 38
SP - 1679
EP - 1682
JO - Neurology
JF - Neurology
IS - 11
ER -